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Die Berechnung der individuellen Insulin Sensitivität ist abhängig von der verwendeten Messmethodik (2010)

Schöndorf, T. ; Musholt, P. B. ; Mohr, T. A. ; Diessel, S. ; Forst, T. ; Konrad, T. ; Backus, J. ; Löbig, M. ; Pfützner, A.

PTEN mutations do not cause nuclear β-catenin accumulation in endometrial carcinomas (2004)

Wappenschmidt, B. ; Wardelmann, E. ; Gehrig, A. ; Schöndorf, T. ; Maass, N. ; Bonatz, G. ; Gassel, A.-M. ; Pietsch, T. ; Mallmann, P. ; Weber, B.H.F. ; Schmutzler, R. K.

A new targeting approach for breast cancer gene therapy using the Heparanase promoter (2005)

Breidenbach, Martina ; Rein, Daniel T. ; Schöndorf, Thomas ; Khan, Kiran N. ; Herrmann, Isabell ; Schmidt, Torsten ; Reynolds, Paul N. ; Vlodavsky, Israel ; Haviv, Yosef S. ; Curiel, David T.

Review of the Pleiotropic Effects of Peroxisome Proliferator-Activated Receptor γ Agonists on Platelet Function (2007)

Borchert, M. ; Schöndorf, T. ; Lübben, G. ; Forst, T. ; Pfützner, A.

Die Relaxinexpression korreliert mit Änderungen der Fibrinogenspiegel bei Patientinnen mit Typ-2-Diabetes unter oraler antidiabetischer Therapie (2007)

Borchert, M. ; Pfützner, A. ; Breidenbach, M. ; Forst, T. ; Luebben, G. ; Hohberg, C. ; Armbruster, F. P. ; Schöndorf, T.

Comparative evaluation of the stada glucocheck monitoring device (2008)

Weise, A. ; Scherze, J. ; Grenningloh, M. ; Schöndorf, T. ; Hauth, W. ; Lipfert, B. ; Forst, T. ; Pfützner, A.

Fixkombination aus Pioglitazon und Metformin verbessert metabolisches Markerprofil (2010)

Schöndorf, T. ; Pfützner, A. ; Fors, T. ; Schlaeger, C. ; Karagiannis, E.

Changes in insulin resistance and cardiovascular risk induced by PPARgamma activation have no impact on RBP4 plasma concentrations in nondiabetic patients (2009)

Pfützner, A. ; Schöndorf, T. ; Hanefeld, M. ; Lübben, G. ; Kann, P. H. ; Karagiannis, E. ; Wilhelm, B. ; Forst, T.

Dysregulation of protein kinase C activity in chemoresistant metastatic breast cancer cells (2004)

Schöndorf, Thomas ; Hoopmann, Markus ; Breidenbach, Martina ; Rein, Daniel T. ; Göhring, Uwe-Jochen ; Becker, Martina ; Mallmann, Peter ; Kurbacher, Christian M.

This study was performed to evaluate the role of protein kinase C (PKC) activity in the development of chemoresistance in clinical breast cancer cells. To simulate the clinical situation, native tumor cells derived from 10 patients with advanced breast cancer were brought into short-term cultures, and treated with anthracyclines (doxorubicin, mitoxantrone), paclitaxel and combinations, respectively. After 3 days of incubation, we determined total PKC activity relative to each control incubated with blank medium.

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